Cirrhosis or chronic liver disease can cause irreversible injuries or injuries in the liver. Progression of chronic liver failure is slower than acute liver failure, but the outlook is worse. Liver cells are slowly damaged, and the liver slowly becomes dysfunctional.
As with albumin and blood clotting factors, the liver's main ingredients are slowly reduced, and toxic substances are normally transformed into non-toxic substances and multiplied by the liver. Portal hypertension (increased pressure in the main liver vein) and bleeding in the digestive system occur when the injuries block the blood flow in the liver. Cirrhosis of the liver can occur for many reasons.
Cirrhosis is usually seen in America and Europe due to high alcohol consumption. Alcohol-induced liver disease occurs when the proportion of fat in liver cells increases. The gradual progression of the death of these cells leads to inflammation of the liver caused by alcohol 5, and injury.
The proportion of alcohol causing damage to the liver varies from person to person. In general, a small amount of alcohol taken on a daily basis may result in women, and then men may have cirrhosis.
While signs of chronic liver failure can not be observed throughout the years, the liver gradually becomes ineffective, progressively injured and ineffective due to these injuries. General symptoms are fatigue, weakness, loss of appetite and weight loss. Mild jaundice is also among the symptoms. The reason for easy bleeding and bruising is that the blood clot needed by the proteins is not produced in the proper amount.
Your skin can scratch because of the increased toxins. You can be more sensitive to the effects of many drugs cleared by the liver. Toxins start to accumulate and cause mental changes. In the later stages of the disease, yellowing or gallstones may occur depending on the skin.
Also, in the main vein of the liver, high pressure (door veins) that occur due to wound formation causes swelling of veins in the food tube and the veins.
These swollen veins, called varicose veins, can be explosive and life-threatening and become fragile.
With the level of albinus (the main protein produced by the liver) dropping steadily, the carcasses leak into some tissues and cause them to swell, especially in the stools. It creates. The muscles begin to shrink and the arms and thighs are examined.
Doctors are diagnosed according to disease symptoms and physical examinations. Loss of appetite, nausea and some physical irregularities constitute this indication. In the initial stages of cirrhosis, only the liver expands; but in later stages the liver becomes narrowed and becomes molten.
Doctors detect the shape and size of the liver through physical examinations.
In such examinations, the underlying causes of liver failure are; jaundice can be revealed by clues such as spider-blooded thin blood vessels on the skin and uncontrollable hand tremors. Blood tests reveal that the liver is unable to produce essential substances in sufficient proportions, that toxins (such as ammonia) increase blood levels, or that bile ducts become blocked due to injuries. This is an additional strong evidence of the diagnosis of chronic liver failure.
Diagnosis of cirrhosis may also occur by imaging the liver, and biopsy may be used to diagnose cirrhosis.